IRIS
VILLA, VALENTINA
 Distribuzione geografica
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Continente #
EU - Europa 6.816
AS - Asia 250
SA - Sud America 50
NA - Nord America 19
AF - Africa 4
Continente sconosciuto - Info sul continente non disponibili 1
Totale 7.140
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Nazione #
IT - Italia 6.811
SG - Singapore 107
CN - Cina 69
VN - Vietnam 49
BR - Brasile 31
HK - Hong Kong 13
US - Stati Uniti d'America 12
EC - Ecuador 6
MX - Messico 6
AR - Argentina 5
ID - Indonesia 5
CO - Colombia 3
BO - Bolivia 2
PH - Filippine 2
PK - Pakistan 2
VE - Venezuela 2
ZA - Sudafrica 2
AL - Albania 1
BD - Bangladesh 1
BG - Bulgaria 1
DE - Germania 1
GA - Gabon 1
JP - Giappone 1
KE - Kenya 1
PL - Polonia 1
RU - Federazione Russa 1
SY - Repubblica araba siriana 1
TT - Trinidad e Tobago 1
UY - Uruguay 1
XK - ???statistics.table.value.countryCode.XK??? 1
Totale 7.140
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Città #
Genova 3.952
Genoa 1.785
Rapallo 554
Vado Ligure 501
Singapore 47
Bordighera 19
Beijing 17
Ho Chi Minh City 17
Hong Kong 13
Ashburn 9
Hanoi 6
Da Nang 4
Tianjin 4
Haiphong 3
Hải Dương 3
Mexico City 3
Quito 3
Biên Hòa 2
Jakarta 2
Johannesburg 2
La Paz 2
Medellín 2
Ninh Bình 2
São Paulo 2
Araranguá 1
Arraias 1
Babahoyo 1
Barcelona 1
Berazategui 1
Boa Vista 1
Brasília 1
Bulungan 1
Bắc Ninh 1
Caloocan City 1
Camaçari 1
Canguçu 1
Cao Lanh 1
Couva 1
Curitiba 1
Damascus 1
Diamantina 1
Guadalajara 1
Guadalupe 1
Guayaquil 1
Huế 1
Isidro Casanova 1
Itaguaí 1
Ituiutaba 1
Ivinhema 1
Juazeiro do Norte 1
Karachi 1
La Plata 1
Lagoa Santa 1
Libreville 1
Manila 1
Materlândia 1
Mexicali 1
Mogi das Cruzes 1
Monte Alegre de Sergipe 1
Montevideo 1
Nairobi 1
Nanjing 1
Nha Trang 1
North Highlands 1
Nova Iguaçu 1
Nārāyanganj 1
Palmeiras de Goiás 1
Peshawar 1
Peçanha 1
Pouso Alegre 1
Pristina 1
Quixeramobim 1
Quận Ninh Kiều 1
Ribeirão Preto 1
Riobamba 1
Rivera 1
San Jose 1
Santa Clara 1
Santo André 1
Santo Tomé 1
Sapezal 1
Sobradinho 1
Sofia 1
São José do Ouro 1
Tasikmalaya 1
Termas de Río Hondo 1
Thái Bình 1
Tirana 1
Tokyo 1
Umuarama 1
Uruaçu 1
Usinsk 1
Vitória de Santo Antão 1
Vĩnh Tường 1
Warsaw 1
Totale 7.025
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Nome #
Amyloid precursor protein modulates ERK1/2 signaling 223
Characterization of the pro-apoptotic intracellular mechanisms induced by a toxic conformer of the recombinant human prion protein fragment 90-231 217
Cellular prion protein controls stem cell-like properties of human glioblastoma tumor-initiating cells 217
Calcium binding promotes Prion Protein fragment 90-231 conformational change toward a membrane destabilizing and cytotoxic structure 215
Peptide Receptor Targeting in Cancer: The Somatostatin Paradigm 204
Basic fibroblast growth factor activates endothelial nitric-oxide synthase in CHO-K1 cells via the activation of ceramide synthesis 199
Apoptotic cell death and impairment of L-type voltage sensitive calcium channel activity in rat cerebellar granule cells treated with the prion protein fragment 106-126 196
Dual modulation of ERK1/2 and p38 MAP kinase activities induced by minocycline reverses the neurotoxic effects of the prion protein fragment 90-231 192
Amino-terminally truncated prion protein PrP90-231 induces microglial activation in vitro 186
Pharmacological activation of autophagy favors the clearing of intracellular aggregates of misfolded prion protein peptide to prevent neuronal death 178
Celecoxib Inhibits Prion Protein 90-231-Mediated Pro-inflammatory Responses in Microglial Cells 175
Different Molecular Mechanisms Mediate Direct or Glia-Dependent Prion Protein Fragment 90–231 Neurotoxic Effects in Cerebellar Granule Neurons 175
Different structural stability and toxicity of PrPARR and PrPARQ sheep prion protein variants 166
Emerging targets in pituitary adenomas: role of the CXCL12/CXCR4-R7 system 166
Contribution of two conserved glycine residues to fibrillogenesis of the 106-126 prion protein fragment. Evidence that a soluble variant of the 106-126 peptide is neurotoxic 164
Canine osteosarcoma cell lines contain stem-like cancer cells: Biological and pharmacological characterization 163
Human PrP90-231-induced cell death is associated with intracellular accumulation of insoluble and protease-resistant macroaggregates and lysosomal dysfunction 160
Novel celecoxib analogues inhibit glial production of prostaglandinE2, nitric oxide, and oxygen radicals reverting the neuroinflammatoryresponses induced by misfolded prion protein fragment 90-231 or lipopolysaccharide 160
Excitotoxicity Through NMDA Receptors Mediates Cerebellar Granule Neuron Apoptosis Induced by Prion Protein 90-231 Fragment 158
The chemokine SDF1/CXCL12: a novel autocrine/paracrine factor involved in pituitary adenoma development 156
ERK1/2 and p38 MAP kinases control prion protein fragment 90-231-induced astrocyte proliferation and microglia activation 150
Conformation dependent pro-apoptotic activity of the recombinant human prion protein fragment 90-231 149
Prion protein fragment 106-126 induces a p38 MAP kinase-dependent apoptosis in SH-SY5Y neuroblastoma cells independently from the amyloid fibril formation 147
The phosphotyrosine phosphatase PTPh mediates somatostatin inhibition of glioma proliferation via the dephosphorylation of ERK1/2 147
ACTIVATION OF AUTOPHAGY PATHWAY AS NEUROPROTECTIVE MECHANISM AGAINST AMYLOIDOGENIC PRION PEPTIDE 145
Role of prion protein aggregation in neurotoxicity 142
Expression in E. coli and purification of recombinant fragments of wild type and mutant human prion protein 141
Efficacy of novel acridine derivatives in the inhibition of hPrP90-231 prion protein fragment toxicity 140
Recombinant human prion protein fragment 90-231, a useful model to study prion neurotoxicity 139
p38 MAP Kinase mediates the cell death induced by PrP106-126 in the SH-SY5Y neuroblastoma cells 137
Celecoxib, a COX-2 inhibitor, blocks microglial activation induced by a neurotoxic conformer of hPrP90-231 137
High hydrophobic amino acid exposure is responsible of the neurotoxic effects induced by E200K or D202N disease-related mutations of the human prion protein 137
Aminoterminally truncated human Prion protein fragment 90-231 forms intracellular aggregates and causes lysosomal loss of impermeability 134
Addressing thermal and mechanical stability of human prion protein with single molecule and ensemble measurements 133
Psychological treatments and psychotherapies in the neurorehabilitation of pain: Evidences and recommendations from the italian consensus conference on pain in neurorehabilitation 130
Molecular mechanisms involved in the aggregation and fibril formation of the recombinant human prion fragment PrP90-231 118
Somatostatin inhibits tumor angiogenesis and growth via somatostatin receptor 3 (SSTR3) mediated regulation of endothelial NOS and MAP kinase activities” 114
Nitric oxide production stimulated by the basic fibroblast growth factor requires the synthesis of ceramide 112
Neurodegeneration in Alzheimer disease: role of amyloid precursor protein and presenilin 1 intracellular signaling 112
Intracellular accumulation of a mild-denatured monomer of the human PrP fragment 90-231 (hPrP90-231), as possible mechanism of its neurotoxic effects 110
Study of the Molecular Mechanism Responsible of the Conformational Changes Induced by E200K and D202N Pathological PrP Mutation 101
Expression of somatostatin receptors mRNA in human meningiomas and their implication in in vitro antiproliferative activity 101
Prion protein fragment 90-231 induces microglia activation: role of ERK1/2 and p38 MAP kinases in nitric oxide production and CCL5 secretion 99
Molecular mechanisms mediating the neuroprotective effects of quinacrine and minocycline on cell death induced by the prion protein fragment 90-231 (hPrP90-231) 97
Study of the molecular mechanisms responsible of the transition of the prion protein fragment 90-231 in a neurotoxic conformation. 90
Somatostatinergic control of Kaposi’s sarcoma growth through the inhibition of angiogenesis 88
Neurotoxicity of the recombinant prion protein fragment 90-231 in SH-SY5Y cells is dependent on its conversion in beta-structured-isoform 79
Somatostatin receptor expression in patients with stromal tumors of the gastrointestinal tract 73
Somatostatin receptor expression in stromal tumors of the gastrointestinal 66
Characterization of the intracellular mechanisms mediating somatostatin and lanreotide inhibition of DNA synthesis and growth hormone release from dispersed human GH-secreting pituitary adenoma cells in vitro 64
The activation of the phosphotyrosine phosphatase eta is responsible for the somatostatin inhibition of PC Cl3 thyroid cell proliferation 54
Totale 7.256
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Categoria #
all - tutte 22.249
article - articoli 20.754
book - libri 0
conference - conferenze 1.182
curatela - curatele 0
other - altro 0
patent - brevetti 0
selected - selezionate 0
volume - volumi 313
Totale 44.498
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Totale Lug Ago Sett Ott Nov Dic Gen Feb Mar Apr Mag Giu
2020/2021216 0 0 0 0 0 30 31 45 24 38 22 26
2021/2022672 16 75 35 68 20 52 61 149 20 54 32 90
2022/2023648 48 43 5 64 110 116 1 54 94 5 101 7
2023/2024346 22 33 7 48 22 87 28 20 17 6 24 32
2024/20251.197 32 68 30 117 165 123 92 232 39 44 122 133
2025/2026998 243 51 252 184 244 24 0 0 0 0 0 0
Totale 7.256