IRIS
BONIFACINO, TIZIANA
 Distribuzione geografica
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Continente #
EU - Europa 13.931
AS - Asia 148
SA - Sud America 19
NA - Nord America 3
Totale 14.101
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Nazione #
IT - Italia 13.930
CN - Cina 120
BR - Brasile 15
VN - Vietnam 12
SG - Singapore 10
ID - Indonesia 4
US - Stati Uniti d'America 3
AR - Argentina 2
CL - Cile 1
CO - Colombia 1
GB - Regno Unito 1
HK - Hong Kong 1
KW - Kuwait 1
Totale 14.101
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Città #
Genova 6.178
Genoa 5.251
Vado Ligure 1.295
Rapallo 1.170
Beijing 52
Bordighera 34
Hanoi 5
Ashburn 3
Ho Chi Minh City 3
Buenos Aires 2
Curitiba 2
Singapore 2
São Paulo 2
Alegre 1
As Sālimīyah 1
Barra do Corda 1
Bonito 1
Campos dos Goytacazes 1
Estación Colina 1
Guaramirim 1
Hangzhou 1
Hong Kong 1
Hải Dương 1
Kisaran 1
Medan 1
Milan 1
Nha Trang 1
Pekanbaru 1
Pontal do Paraná 1
Porto Alegre 1
Portão 1
Santo Antônio da Patrulha 1
Thái Bình 1
Três Barras do Paraná 1
Turin 1
Valparaíso de Goiás 1
Totale 14.022
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Nome #
Acute stress increases depolarization-evoked glutamate release and presynaptic SNARE complex accumulation in prefrontal/frontal cortex. The dampening action of antidepressants. 212
Genetic inactivation of mGlu5 receptor improves motor coordination in the Grm1crv4 mouse model of SCAR13 ataxia 203
In-vivo effects of knocking-down metabotropic glutamate receptor 5 in the SOD1G93A mouse model of amyotrophic lateral sclerosis. 197
In-vivo genetic ablation of metabotropic glutamate receptor type 5 slows down disease progression in the SOD1G93A mouse model of amyotrophic lateral sclerosis 194
Blockade of stress-induced increase of glutamate release in the rat prefrontal/frontal cortex by agomelatine involves synergy between melatonergic and 5-HT2C receptor-dependent pathways 183
Characterization of the Mitochondrial Aerobic Metabolism in the Pre- and Perisynaptic Districts of the SOD1G93A Mouse Model of Amyotrophic Lateral Sclerosis 180
Altered mechanisms underlying the abnormal glutamate release in amyotrophic lateral sclerosis at a pre-symptomatic stage of the disease 175
Altered Mechanisms Underlying the Abnormal Glutamate Release in Amyotrophic Lateral Sclerosis at a Pre-Symptomatic Stage of the Disease. 167
Exocytosis regulates trafficking of GABA and glycine heterotransporters in spinal cord glutamatergic synapses: a mechanism for the excessive heterotransporter-induced release of glutamate in experimental amyotrophic lateral sclerosis. 165
The endocannabinoid system in rat gliosomes and its role in the modulation of glutamate release 160
Colocalization of neurotransmitter transporters on the plasma membrane of the same nerve terminal may reflect cotransmission. 152
Acute stress rapidly increases the readily releasable pool of glutamate vesicles in prefrontal and frontal cortex through non-genomic action of corticosterone 151
Abnormal exocytotic release of glutamate in a mouse model of amyotrophic lateral sclerosis 151
Enhanced Function and Overexpression of Metabotropic Glutamate Receptors 1 and 5 in the Spinal Cord of the SOD1G93A Mouse Model of Amyotrophic Lateral Sclerosis during Disease Progression 149
Chronic mild stress induces anhedonic behavior and changes in glutamate release, BDNF trafficking and dendrite morphology only in stress vulnerable rats. The rapid restorative action of ketamine 148
Enriched experience and recovery from amblyopia in adult rats: impact of motor, social and sensory components. 146
Knocking down metabotropic glutamate receptor 1 improves survival and disease progression in the SOD1G93A mouse model of amyotrophic lateral sclerosis 143
Group I metabotropic glutamate autoreceptors induce abnormal glutamate exocytosis in a mouse model of amyotrophic lateral sclerosis 141
Chronic treatment with agomelatine or venlafaxine reduces depolarization-evoked glutamate release from hippocampal synaptosomes 140
Abnormal exocytotic release of glutamate in a mouse model of amyotrophic lateral sclerosis. 136
Altered glucose catabolism in the presynaptic and perisynaptic compartments of SOD1G93A mouse spinal cord and motor cortex indicates that mitochondria are the site of bioenergetic imbalance in ALS 136
Metabotropic glutamate receptor 5 as a potential target in ALS 135
Differential expression of metabotropic glutamate and GABA receptors at neocortical glutamatergic and GABAergic axon terminals 134
Fluoxetine in adulthood normalizes GABA release and rescues hippocampal synaptic plasticity and spatial memory in a mouse model of Down Syndrome. 132
Abnormal and precocious exocytotic glutamate release in a mouse model of amyotrophic lateral sclerosis 132
Intravenous Mesenchymal Stem Cells Improve Survival and Motor Function in Experimental Amyotrophic Lateral Sclerosis 132
Glutamate release from astrocytic gliosomes under physiological and pathological conditions 127
Intravenous mesenchymal stem cells improve survival and motor functions in symptomatic mutant SOD1/G93A mice 125
Role of calpain-1 in the early phase of experimental ALS 125
mGluR1 and mGluR5 knock-down improves survival, motor skills and disease progression in amyotrophic lateral sclerosis mouse model 121
Acute stress rapidly increases the readily releasable pool of glutamate vesicles in prefrontal and frontal cortex through non-genomic action of corticosterone. 120
Abnormal glutamate release in a mouse model of amyotrophic lateral sclerosis 120
Amyotrophic lateral sclerosis and excitotoxicity: role of Group I metabotropic glutamate receptors 118
Acute stress and corticosterone increase the readily releasable pool of glutamatergic vesicles in rat synaptosomes of prefrontal/frontal cortex via mineralocorticoid and glucocorticoid receptors 117
Astrocyte contribution to the excessive glutamate release in the spinal cord of the SOD1G93A mouse model of amyotrophic lateral sclerosis 117
Targeting Group I Metabotropic Glutamate Receptors in Amyotrophic Lateral Sclerosis 116
Abnormal glutamate release in a mouse model of amyotrophic lateral sclerosis 116
GroupI metabotropic glutamate autoreceptors induce abnormal glutamate exocytosis in a mouse model of amyotrophic lateral sclerosis 114
Group I metabotropic glutamate receptors tune excitotoxicity in amyotrophic lateral sclerosis 114
Stress and corticosterone increase the readily releasable pool of glutamate vesicles in synaptic terminals of prefrontal and frontal cortex 114
GROUP I METABOTROPIC GLUTAMATE RECEPTORS AND NEUROTOXICITY IN AMYOTROPHIC LATERAL SCLEROSIS 113
Effect of miRNAs shuttled by exosomes derived from primed-MSCs on activated-microglia phenotype and possible implication in amyotrophic lateral sclerosis. 113
In vitro Evidence of Abnormal Glutamate to Glutamine Conversion in Astrocytes harvested from a Mouse Model of Amyotrophic Lateral Sclerosis: a potential Experimental Application of N-13-ammonia 112
Synaptic mechanisms underlying the excessive and precocious glutamate release in the spinal cord of sod1g93a mice. 111
Knocking-down mGluR1 and mGluR5 in the SOD1/G93A mouse model of amyotrophic lateral sclerosis ameliorates survival and disease progression 111
Increased [3H]D-aspartate release and changes in glutamate receptor expression in the hippocampus of the mnd mouse. 110
Acute ketamine restores deficits in glutamate release and related molecular mechanisms induced by chronic mild stress in vulnerable rats 110
Abnormal Upregulation of GPR17 Receptor Contributes to Oligodendrocyte Dysfunction in SOD1 G93A Mice 110
Knocking-down mGlu1 receptors prolongs survival and ameliorates disease progression in the SOD1/G93A mouse model of ALS. 109
Altered Mechanisms Underlying the Abnormal Glutamate Release in Amyotrophic Lateral Sclerosisat a Pre-Symptomatic Stage of the Disease. 109
Ketamine modulates glutamate release, BDNF trafficking and dendrite morphology in rats vulnerable to chronic mild stress 107
Abnormal glutamate release induced by group I metabotropic glutamate receptors in experimental ALS. 103
Altered synaptic mechanisms and excessive glutamate release in amyotrophic lateral sclerosis 102
Genetic and pharmacological effects of mGlu5 receptor blockade in the SOD1G93A mouse model of amyotrophic lateral sclerosis 102
A multistationary loop model of ALS unveils critical molecular interactions involving mitochondria and glucose metabolism 102
miRNAs shuttled by exosomes derived from primed-MSCs affect the activated-microglia phenotype and possibly support the therapeutic effects of MSC administration in SOD1G93A mice 101
Targeting group i metabotropic glutamate receptors in experimental als. 101
Alterations of Synaptic Mechanisms and Excessive Glutamate Release in the Spinal Cord of SOD1G93A Mice 101
In-vivo therapeutic effects of extracellular vesicles derived from mesenchymal stem after systemic administration on ALS mice 100
Fluoxetine treatment promotes functional recovery in a rat model of cervical spinal cord injury 100
The Modulation of Glutamate Release by Group I Metabotropic Glutamate Auto-Receptors in ALS 100
Acute stress rapidly increases the readily releasable pool of glutamate vesicles in prefrontal and frontal cortex through corticosterone non-genomic action 100
Knocking down mglu1 receptors prolongs survival and improves symptomatology in a mouse model of amyotrophic lateral sclerosis 99
Intravenous mesenchymal stem cells administration induces survival and symptom amelioration in mutant SOD1 G93A mice 99
Knocking-down Group I metabotropic glutamate receptors ameliorates survival and disease progression in SOD1G93A mice. 98
Group I metabotropic glutamate receptors modulate glutamate exocytosis in a mouse model of als 98
In-vitro activation of GAT1 transporters expressed in spinal cord gliosomes stimulates glutamate release that is abnormally elevated in the SOd1/G93A(+) mouse model of amyotrophioc lateral sclerosis. 98
Group I metabotropic glutamate receptors in ALS: knocking-down mGlu1 receptors ameliorates survival and disease progression in SOD1/G93A mice. 97
Genetic inactivation of metabotropic glutamate 5 (mGlu5) receptor ameliorates motor coordination in the Grm1crv4 mouse model of SCAR13 ataxia 97
Alterations of Synaptic Mechanisms and Excessive Glutamate Release in the Spinal Cord of SOD1G93A Mice 97
Non conventional activity of the endocannabinoid system at glutamate release in rat cortical astrocytes 96
Mesemchymal stem cells systemically administered in symptomatic mutant SOD1-G93A mice ameliorate survival and motor abilities 96
The modulation of Glutamate release by Group I metabotropic Glutamate auto-receptors in ALS 96
GLUTAMATE NEUROTRANSMISSION IS MODIFIED IN THE SOD1/G93A MOUSE MODEL OF ALS 93
Reducing the expression of mGlu1 and mGlu5 receptors ameliorates survival and disease progression in SOD1G93A mice 93
Group I metabotropic glutamate receptors induce excessive glutamate release in the spinal cord of sod1g93a mice 93
Changes at glutamate tripartite synapses in the prefrontal cortex of a new animal model of resilience/vulnerability to acute stress 93
Molecular mechanisms supporting abnormal glutamate release in the spinal cord of a mouse model of Amyotrophic Lateral Sclerosis. 91
Intravenous mesenchymal stem cells administration induces survival and symptom amelioration in mutant SOD1 G93A mice 91
Inactivation of the Grm5 gene improves motor coordination defects in the Grm1crv4 mouse model of SCAR13 ataxia 90
Altered expression of the GPR17 receptor in the spinal cord of SOD1G93A mice, a model of amyotrophic lateral sclerosis 89
The complex phenotype and function of spinal cord microglia during ALS progression and the impact of metabotropic glutamate receptor type 5 down-regulation in SOD1G93A mice 88
Knocking-down mGluR1 and mGluR5 in SOD1G93A mice ameliorates survival and disease progression 88
Ketamine restores changes in glutamate release, dendrite morphology and BDNF trafficking in the hippocampus of rats vulnerable to chronic mild stress 88
Acute stress increases the readily releasable pool of glutamate vesicles in cortical areas 87
Genetic and pharmacological effects by blocking mGlu5 Receptor in the SOD1G93A Mouse Model of Amyotrophic Lateral Sclerosis 87
In-vitro and in-vivo studies depict metabotropic glutamate receptor 5 as a potential pharmacological target to modulate disease progression in ALS 87
Partial deletion of mGluR1 receptors prolongs life span and ameliorates motor skills and biochemical and cellular parameters in a mouse model of experimental ALS 86
Reducing excitotoxicity knocking down mGluR1: prolonged survival and improved symptomatology of SOD1/G93A mouse model of ALS 86
G93A mutated SOD1-linked murine ALS produces abnormal exocytotic release of glutamate 86
Abnormal and precocious exocytotic release of glutamate in the spinal cord of a mouse model of amyotrophic lateral sclerosis. 86
Excessive glutamate release and undelying synaptic mechanisms in a mouse model of amyotrophic lateral sclerosis 86
Group I metabotropic glutamate receptors in ALS: mGluR1 and mGluR5 autoreceptors induce excessive glutamate release in SOD1/G93A mice. 85
Synaptic Mechanisms Sustaining The Excessive And Precocious Glutamate Release In The Spinal Cord Of SOD1G93A Mice 85
Mechanisms underlying the predictive power of high skeletal muscle uptake of FDG in amyotrophic lateral sclerosis 85
Neuroprotective Effect of AM404 Against NMDA-Induced Hippocampal Excitotoxicity 84
Blocking glutamate mGlu5 receptors with the negative allosteric modulator CTEP improves disease course in SOD1G93A mouse model of amyotrophic lateral sclerosis 84
Acute Ketamine Facilitates Fear Memory Extinction in a Rat Model of PTSD Along With Restoring Glutamatergic Alterations and Dendritic Atrophy in the Prefrontal Cortex 83
Genetic Downregulation of the Metabotropic Glutamate Receptor Type 5 Dampens the Reactive and Neurotoxic Phenotype of Adult ALS Astrocytes 82
Reducing mglur5 expression attenuates the reactive phenotype of astrocytes cultured from the spinal cord of late symptomatic SOD1G93A mice 82
Totale 11.514
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Categoria #
all - tutte 50.336
article - articoli 21.899
book - libri 478
conference - conferenze 27.959
curatela - curatele 0
other - altro 0
patent - brevetti 0
selected - selezionate 0
volume - volumi 0
Totale 100.672
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Totale Lug Ago Sett Ott Nov Dic Gen Feb Mar Apr Mag Giu
2020/2021575 0 0 0 41 59 64 67 89 63 66 56 70
2021/20221.332 21 85 62 127 50 107 83 322 84 126 68 197
2022/20231.440 137 147 21 164 212 201 15 104 214 12 183 30
2023/20241.113 36 124 33 135 80 235 95 43 46 61 74 151
2024/20253.571 189 237 82 240 388 325 302 664 103 159 408 474
2025/20261.895 822 173 435 465 0 0 0 0 0 0 0 0
Totale 14.636